Gene runner

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Gene Runner is a professional application aimed at providing geneticists and biologists with a reliable tool that can help them conduct gene analysis with ease. Are you having trouble uninstalling Gene Runner? Are you looking for a solution that will completely uninstall and remove all of its files from your computer? This guide will provide you with detailed instructions and all the information that you require to remove and uninstall Gene Runner.What usually makes people to uninstall Gene Runner?It seems that there are many users who have difficulty uninstalling programs like Gene Runner from their systems. Some experience issues during uninstallation, whereas other encounter problems after the program is removed.These are the main reasons why Gene Runner is uninstalled by users:The program is not compatible with other installed applications.The program crashes or hangs periodically/frequently.The program is not as good as the user expected.The program is being re-installed (uninstalled and installed again afterwards).The program is regarded by the user and/or some websites as a potentially malicious.The program is not listed in the Windows Settings or Control Panel.After removing the program, some of its related processes still run on the computer.Some traces of the program can still can be found on the computer.Removing process running for a long time and does not come to the end.Possible problems that can arise during uninstallation of Gene RunnerThe program’s built-in uninstaller does not start or does not operate properly.A file required for the uninstallation to complete could not be run.Due to an error, not all of the files were successfully uninstalled.Another process is preventing the program from being uninstalled.There could be other causes why users may not be able to uninstall Gene Runner. An incomplete uninstallation of a program may cause problems, which is why thorough removal of programs is recommended.How to uninstall Gene Runner completely?Method 1: Uninstall Gene Runner with a third-party uninstaller.1Download and install Revo Uninstaller Pro - 30 days fully functional trial version2Start Revo Uninstaller Pro and open the module "Logs Database" 3In the Search field type "Gene Runner" and you will see all logs of "Gene Runner" in the database compatible with your Windows Version.4Select the appropriate log by version from the list and press the "Uninstall" button from the toolbar 5You will see few popping up windows showing the download and import of the log to your Revo Uninstaller Pro and then the main Uninstall dialog that shows the progress of the uninstall of Gene Runner.Method 2: Uninstall Gene Runner via Apps and Features/Programs and Features.1Open the Start Menu and type Apps and Features 2Look for Gene Runner in the list and click on it. The next step is to click on uninstall, so you can initiate the uninstallation. Method

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3: Uninstall Gene Runner with its uninstaller.exe.Most of the computer programs have uninstall.exe or uninst000.exe in their installation folders. You need to execute these files in order to start the uninstallation process. Here are the steps:1Go to the installation folder of Gene Runner. Most of the times it is located in C:\Programs files or C:\Program files(x86)2Locate uninstall.exe or uninst000.exe 3Double click the file to start the uninstallation process.Method 4: Uninstall String with Run1Hold the Windows + R keys to open the Run command. 2 this uninstallation stringMsiExec.exe /I{2C06BB7C-6A69-44C6-9A13-21EB45B17C72}and paste it in the Run command window and click OK.3Follow the instructions to complete the uninstallation. We can help you every time when…you cannot find the program in the Apps & Features listthe program's built-in uninstaller is non-functionalyou have a lot of leftovers slowing down your computer's performanceyou want to batch uninstallmany more things Download nowAvailable logsGene Runner 6.5 on Windows 7 64-bitGene Runner 6.5 on Windows 7 32-bitGene Runner 6.5 on Windows 8.1 64-bitGene Runner 6.5 on Windows 10 64-bitGene Runner 6.5 on Windows 10 32-bitRelated programsNo related products

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Mangrum, Jug McSpaden, Byron Nelson, Gene Sarazen, Horton Smith, Craig Wood player/captain). Once again, the Ryder Cup Team triumphs, 10 to 5.1940s-60: Hearst Tournaments are hosted by North Hills Golf Course from the mid-40s until 1960 when The Detroit Times, the local Hearst newspaper and sponsor, is sold to The Detroit News.1946: Hearst National Junior Championship. In his first national tournament competition, 16-year-old Arnold Palmer is runner-up to MacGregor Hunter.1948: Donald Ross dies April 26, 1948. His 1938 rough drawings of suggestions for changes to the South Course are turned over to Robert Trent Jones once he is hired in 1950 to modernize the course.1949: John Anderson, an Oakland Hills caddy, is the first caddy in the Detroit District to be awarded a college scholarship by the Western Golf Association. 1950: Robert Trent Jones is hired to modernize and strengthen the South Course. He narrows the fairways, eliminates bunkers that are out of play and adds new bunkers to squeeze the tee shot landing zones and the entryways to greens. The course goes from just under 90 bunkers to just over 120. On 15 he replaces the old fairway bunkers with one new one – in the dead center of the fairway driving zone. On 16 he nudges the green onto a new peninsula in the pond to create a more precarious approach shot. He reduces par on eight and 18 to 4 for tournament play. Most penal of all is the unintended consequence of over-seeding the ground around the new bunkers with rye grass to hold everything in place. The rye grows in fast and thick and stubbornly resists letting go of golf balls. In the process of this work, Jones invents the modern championship golf course. After Oakland Hills, Jones is hired to renovate Baltusrol (1952), Olympic (1954), Oak Hill (1956), Southern Hills (1957), and Winged Foot (1958), all in preparation for U. S. Opens. Jones becomes known as the "Open Doctor." 1951: U. S. Open. Defending champion Ben Hogan shoots a 287 (76, 73, 71, 67) to win his third U. S. Open by two strokes. He calls his final round 67 the greatest of his career and calls Oakland Hills "the hardest course I've ever played." At the award ceremony Hogan sums up his feelings with a few words that become more famous than any victory speech in the history of the U. S. Open: "I'm glad I brought this course –this monster—to its knees." Clayton Heafner is second with 289, Bobby Locke third with 291, and Lloyd Mangrum fourth with 293.1952-88: Dorothy Thompson Keller, over a 36 year period, wins 12 club championships and is runner-up 14 times.1955: Michigan Women's Amateur returns to Oakland Hills for a third time. Won by Margaret "Wiffi" Smith, who will win the World Women's Amateur and the North and South Women's Amateur this same year.1960: All Star Golf television with host Jimmy Demaret films matches for three days at Oakland Hills: Bill Collins beats Dick Metz 71 to 78.. Get gene runner manual PDF file for free from our online library. GENE RUNNER MANUAL. Gene Runner Manual Download. Gene Runner Manual Free. Gene Runner Manual Full. Gene

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Common type of non-cancerous growth in the uterusWellness ReportsCat AllergyDog AllergyEmotional EatingNearsightednessSeasonal AllergiesHomocysteine (MTHFR-Related)Exome Sequencing Reports6 reports (covering 55+ conditions ordered by a clinician)Hereditary Cancer ReportCoverage includes the following conditions:APC-associated polyposis (Gene: APC)ATM-associated cancers (Gene: ATM)Juvenile polyposis syndrome (Gene: BMPR1A, SMAD4)Hereditary breast and ovarian cancer (Gene: BRCA1, BRCA2)CHEK2-associated cancers (Gene: CHEK2)Hereditary prostate cancer (Gene: HOXB13)Hereditary paraganglioma-pheochromocytoma syndrome (Gene: MAX, SDHAF2, SDHB, SDHC, SDHD, TMEM127)Multiple endocrine neoplasia type 1 (Gene: MEN1)Lynch syndrome (Gene: MLH1, MSH2, MSH6, PMS2)MUTYH-associated polyposis (Gene: MUTYH)NF2-related schwannomatosis (Gene: NF2)Hereditary breast cancer (Gene: PALB2)Polymerase proofreading-associated polyposis (Gene: POLD1, POLE)PTEN hamartoma tumor syndrome (Gene: PTEN)Retinoblastoma (Gene: RB1)Familial medullary thyroid cancer (Gene: RET)Multiple endocrine neoplasia type 2a (Gene: RET)Multiple endocrine neoplasia type 2b (Gene: RET)Juvenile polyposis/hereditary hemorrhagic telangiectasia syndrome (Gene: SMAD4)Peutz-Jeghers syndrome (Gene: STK11)Li-Fraumeni syndrome (Gene: TP53)Tuberous sclerosis complex (Gene: TSC1, TSC2)Von Hippel-Lindau syndrome (Gene: VHL)Wilms tumor (Gene: WT1)Hereditary Cardiovascular Disease ReportCoverage includes the following conditions:Familial thoracic aortic aneurysm and dissection (Gene: ACTA2, MYH11)Familial hypertrophic cardiomyopathy (Gene: ACTC1, MYBPC3, MYH7, MYL2, MYL3, PRKAG2, TNNI3, TNNT2, TPM1)Familial hypercholesterolemia (Gene: APOB, LDLR, LDLRAP1, PCSK9)Type III hyperlipoproteinemia (Gene: APOE)Dilated cardiomyopathy (Gene: BAG3, DES, FLNC, LMNA, MYH7, RBM20, SCN5A, TNNC1, TNNT2, TTN)Myofibrillar myopathy (Gene: BAG3, DES, FLNC)Long QT syndrome (Gene: CALM1, CALM2, CALM3, KCNH2, KCNQ1, SCN5A, TRDN)Catecholaminergic polymorphic ventricular tachycardia (Gene: CALM1, CALM2, CALM3, CASQ2, RYR2, TRDN)Ehlers-Danlos syndrome, vascular type (Gene: COL3A1)Arrhythmogenic right ventricular cardiomyopathy (Gene: DSC2, DSG2, DSP, PKP2, TMEM43)Dilated cardiomyopathy with wooly hair, palmoplantar keratoderma, and tooth agenesis (Gene: DSP)Marfan syndrome (Gene: FBN1)Brugada syndrome (Gene: SCN5A)Loeys-Dietz syndrome (Gene: SMAD3, TGFB2, TGFB3, TGFBR1, TGFBR2)Hereditary Metabolic Disease ReportCoverage includes the following conditions:Biotinidase deficiency (Gene: BTD)G6PD deficiency (Gene: G6PD)Pompe disease (Gene: GAA)Fabry disease (Gene: GLA)Maturity-onset diabetes of the young (Gene: HNF1A)Ornithine carbamoyltransferase deficiency (Gene: OTC)Hereditary Kidney Disease ReportCoverage includes the following conditions:APOL1-related chronic kidney disease (Gene: APOL1)Autosomal dominant polycystic kidney disease (Gene: PKD1, PKD2)Hereditary Neurological

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Girl's Junior Amateur. Iowan Edith Estabrooks wins final match 1-up over Hope Seignious. At just 15, Estabrooks is the WWGA's youngest girl's junior champion.1936: Prior to the '37 Open, noted golf architect Albert W. Tillinghast is asked to look over the South Course and offer advice for improvements. Tillinghast reports, "This course needs nothing more to prepare it for the Open. What it needs is to be left alone." He also observes, "Oakland Hills is one of the finest golf courses, not alone in this country, but in the world."1936: Joe Dey, Jr., Executive Director of the USGA, approaches Chris Brinke (member and noted amateur golfer) and suggests that Oakland Hills increases par on the eighth hole to 5 and reduces par on 14 to 4, creating a balance of two par 5s per side. The Open is played this way in '37 and the adjustment is made permanent for member play.1937: U. S. Open. Ralph Guldahl shoots a low-scoring record 281 (71, 69, 72, 69) to win first of back-to-back U. S. Opens beating Sam Snead by two strokes in Snead's first Open appearance. Bobby Cruickshank is third with 285 and Harry Cooper fourth with 286. At 7037 yards, this is the first U. S. Open held on a course over 7000 yards long. And this is the last U. S. Open in which players are allowed to carry more than 14 clubs in their bag – Guldahl carries 19 throughout the tournament. Guldahl proves to be golf’s most dominant player from 1937 through ’39.1938: Club hires Donald Ross for suggestions to improve South Course. Preliminary drawings of possible changes to each hole are made. Board decides to put Ross suggestions on hold to cut costs during depression. Only Ross’s suggestions for bunker removal go forward (in 1941 they sod over at least 19 bunkers).1938-59: Margaret Russell, over a 21 year period, wins 15 club championships and is runner-up three times.1938: First swimming pool opens on September 3 thanks to the persistent efforts of member Warren Pease over a ten year period.1939: Women's Western Amateur. Edith Estabrooks returns to Oakland Hills and defeats Ellamae Williams 8 and 6 – a 39 year record-winning margin in the Women's Western Amateur.1940 & 42: With the Ryder Cup suspended during the war, and to help raise money for the Red Cross, Oakland Hills hosts a series of exhibition matches. In 1940, Walter Hagen captains the Ryder Cup Team (Vic Ghezzi, Ralph Guldahl, Jimmy Hines, Dick Metz, Byron Nelson, Henry Picard, Horton Smith, Sam Snead) vs. Gene Sarazen's Challengers (Billy Burke, Harry Cooper, Jimmy Demaret, Ben Hogan, Lawson Little, Ed Oliver, Craig Wood, Gene Sarazen player/captain). The Ryder Cup Team comes out on top 7 to 5. In '42 Hagen changes sides and captains the Challengers (Sam Byrd, Harry Cooper, Ralph Guldahl, Chick Harbert, Clayton Heafner, Lawson Little, Dick Metz, Henry Picard, Jimmy Thomson, Joe Turnesa, Al Watrous) vs. Craig Wood's Ryder Cup Team (Jimmy Demaret, Ed Dudley, Vic Ghezzi, Ben Hogan, Lloyd

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The FANCC gene; relevant for Ashkenazi Jewish descentGRACILE Syndrome1 variant in the BCS1L gene; relevant for Finnish descentGaucher Disease Type 13 variants in the GBA (also known as GBA1) gene; relevant for Ashkenazi Jewish descentGlycogen Storage Disease Type Ia1 variant in the G6PC gene; relevant for Ashkenazi Jewish descentGlycogen Storage Disease Type Ib2 variants in the SLC37A4 geneHereditary Fructose Intolerance4 variants in the ALDOB gene; relevant for European descentLeigh Syndrome, French Canadian Type1 variant in the LRPPRC gene; relevant for French Canadian descentLimb-Girdle Muscular Dystrophy Type 2D1 variant in the SGCA geneLimb-Girdle Muscular Dystrophy Type 2E1 variant in the SGCB gene; relevant for Amish descentLimb-Girdle Muscular Dystrophy Type 2I1 variant in the FKRP geneMCAD Deficiency4 variants in the ACADM gene; relevant for European descentMaple Syrup Urine Disease Type 1B2 variants in the BCKDHB gene; relevant for Ashkenazi Jewish descentMucolipidosis Type IV1 variant in the MCOLN1 gene; relevant for Ashkenazi Jewish descentNeuronal Ceroid Lipofuscinosis (CLN5-Related)1 variant in the CLN5 gene; relevant for Finnish descentNeuronal Ceroid Lipofuscinosis (PPT1-Related)3 variants in the PPT1 gene; relevant for Finnish descentNiemann-Pick Disease Type A3 variants in the SMPD1 gene; relevant for Ashkenazi Jewish descentNijmegen Breakage Syndrome1 variant in the NBN geneNonsyndromic Hearing Loss and Deafness, DFNB1 (GJB2-Related)8 variants in the GJB2 gene; relevant for many ethnicities, including Ashkenazi Jewish, East/Southeast Asian, European, and Ghanaian descent. May also be relevant for Hispanic/Latino, Northern African/Middle Eastern, and South Asian descentPendred Syndrome and DFNB4 Hearing Loss (SLC26A4-Related)6 variants in the SLC26A4 genePhenylketonuria and Related Disorders23 variants in the PAH gene; relevant for Irish, Northern European descentPompe Disease5 variants in the GAA gene; relevant for African/African American descent; variants also common in European descentPrimary Hyperoxaluria Type 21 variant in the GRHPR genePyruvate Kinase Deficiency1 variant in the PKLR geneRhizomelic Chondrodysplasia Punctata Type 11 variant in the PEX7 geneSalla Disease1 variant

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Market Closed - Nasdaq Other stock markets 04:00:00 2025-03-21 pm EDT 5-day change 1st Jan Change 1.640 USD -3.53% -4.65% -5.20% Taysha Gene Therapies : Files for Up to $350 Million Mixed Securities Shelf October 06, 2021 at 07:35 am EDT © MT Newswires - 2021 Taysha Gene Therapies, Inc. Reports Earnings Results for the Full Year Ended December 31, 2024 Feb. 26 CI Taysha Gene Therapies, Inc., 2024 Earnings Call, Feb 26, 2025 Feb. 26 Taysha Gene Therapies, Inc.(NasdaqGS:TSHA) added to S&P Biotechnology Select Industry Index Dec. 22 CI Taysha Gene Therapies, Inc., Q3 2024 Earnings Call, Nov 13, 2024 Nov. 13 Earnings Flash (TSHA) TAYSHA GENE THERAPIES Posts Q3 Revenue $1.79M Nov. 13 MT Taysha Gene Therapies, Inc. Reports Impairment of Long-Lived Assets for the Third Quarter Ended September 30, 2024 Nov. 13 CI Taysha Gene Therapies, Inc. Reports Earnings Results for the Third Quarter and Nine Months Ended September 30, 2024 Nov. 13 CI Taysha Gene Therapies, Inc.(NasdaqGS:TSHA) added to S&P Global BMI Index 24-09-22 CI Certain Warrants of Taysha Gene Therapies, Inc. are subject to a Lock-Up Agreement Ending on 26-AUG-2024. 24-08-25 CI Certain Pre-funded Warrants of Taysha Gene Therapies, Inc. are subject to a Lock-Up Agreement Ending on 26-AUG-2024. 24-08-25 CI Certain Restricted Stock Units of Taysha Gene Therapies, Inc. are subject to a Lock-Up Agreement Ending on 26-AUG-2024. 24-08-25 CI Certain Stock Options of Taysha Gene Therapies, Inc. are subject to a Lock-Up Agreement Ending on 26-AUG-2024. 24-08-25 CI Certain Common Stock of Taysha Gene Therapies, Inc. are subject to a Lock-Up Agreement Ending on 26-AUG-2024. 24-08-25 CI Earnings Flash (TSHA) TAYSHA GENE THERAPIES Reports Q2 Revenue $1.1M, vs. Street Est of $2.6M 24-08-12 MT Taysha Gene Therapies, Inc., Q2 2024 Earnings Call, Aug 12, 2024 24-08-12 Taysha Gene Therapies, Inc. Reports Earnings Results for the Second Quarter and Six Months Ended June 30, 2024 24-08-12 CI Taysha Gene Therapies Insider Bought Shares Worth $2,999,999, According to a Recent SEC Filing 24-07-01 MT Piper Sandler Cuts Price Target on Taysha Gene Therapies to $7 From $9, Maintains Overweight Rating 24-07-01 MT Taysha Gene Therapies,. Get gene runner manual PDF file for free from our online library. GENE RUNNER MANUAL. Gene Runner Manual Download. Gene Runner Manual Free. Gene Runner Manual Full. Gene

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For a form of nerve and heart damage3 variants in the TTR gene; relevant for African American, West African, Portuguese, Brazilian, Northern Swedish, Japanese, Irish, British descentHereditary Hemochromatosis (HFE‑Related)Genetic risk for iron overload2 variants in the HFE gene; relevant for European descentHereditary ThrombophiliaGenetic risk for harmful blood clots2 variants in the F2 and F5 genes; relevant for European descentLate-Onset Alzheimer's DiseaseGenetic risk for a form of dementia1 variant in the APOE gene; variant found and studied in many ethnicitiesMUTYH-Associated PolyposisGenetic risk for a specific colorectal cancer syndrome2 variants in the MUTYH gene; relevant for Northern European descentParkinson's DiseaseGenetic risk for a form of movement impairment2 variants in the LRRK2 and GBA genes; relevant for European, Ashkenazi Jewish, North African Berber descentSee sample report - Health PredispositionSee sample report - Health PredispositionCarrier Status Reports*45+ reportsARSACS1 variant in the SACS gene; relevant for French Canadian descentAgenesis of the Corpus Callosum with Peripheral Neuropathy1 variant in the SLC12A6 gene; relevant for French Canadian descentAutosomal Recessive Polycystic Kidney Disease3 variants in the PKHD1 geneBeta Thalassemia and Related Hemoglobinopathies10 variants in the HBB gene; relevant for Sardinian, Cypriot, Italian/Sicilian, Greek descentBloom Syndrome1 variant in the BLM gene; relevant for Ashkenazi Jewish descentCanavan Disease3 variants in the ASPA gene; relevant for Ashkenazi Jewish descentCongenital Disorder of Glycosylation Type 1a (PMM2-CDG)2 variants in the PMM2 gene; relevant for Ashkenazi Jewish, Danish descentCystic Fibrosis29 variants in the CFTR gene; relevant for Ashkenazi Jewish, European, Hispanic/Latino descentD-Bifunctional Protein Deficiency2 variants in the HSD17B4 geneDihydrolipoamide Dehydrogenase Deficiency1 variant in the DLD gene; relevant for Ashkenazi Jewish descentFamilial Dysautonomia1 variant in the ELP1 gene; relevant for Ashkenazi Jewish descentFamilial Hyperinsulinism (ABCC8-Related)3 variants in the ABCC8 gene; relevant for Ashkenazi Jewish descentFamilial Mediterranean Fever7 variants in the MEFV gene; relevant for Arab, Armenian, Sephardic Jewish, Turkish descentFanconi Anemia Group C3 variants in

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Seekers, and Daughter of the Mind. Gene’s last appearance was in 1980 in the miniseries Scruples.Exciting Facts about Gene TierneyGene’s first-ever role on Broadway in What a Life! was just to carry a water bucket across the stage. Tierney struggled with manic depression and was treated in several places like the Institute of Living in Hartford, Connecticut, and Menninger Clinic in Topeka, Kansas. The actress had to give up her lead role in the film Holiday for Lovers because of her poor mental health at the time. Gene Tierney’s autobiography, Self-Portrait, was released in 1979.What face shape does Gene Tierney have?Gene Tierney had a gorgeous heart-shaped face with sharp and contoured cheekbones.What is Gene Tierney’s hair color?Gene Tierney mostly sported short, curled, dark brown hair during her career.What color eyes does Gene Tierney have?Gene had unique and captivating green-gray eyes, which added an appealing charm to her face.Did Gene Tierney win the oscar?Gene Tierney was nominated for an Oscar for Best Actress for the 1945 film Leave Her to Heaven, but she didn’t win the award.Did Gene Tierney play in The Mating Season?Yes, Gene Tierney played the role of Maggie Carleton McNulty in the 1960 rom-com film, The Mating Season.Did Gene Tierney go to school?Yes, Gene Tierney attended different schools as a kid. She went to St. Margaret’s School while growing up in Westport, the Unquowa School in Fairfield, Brillantmont International School in Lausanne, and Miss Porter’s School in Connecticut.Gene Tierney was known for her flawless beauty and refined talent. Having graced over 40 films throughout her career and receiving critical acclaim, she remains a beloved figure in Hollywood history. Lana Turner is another acting icon who graced Hollywood with her 50-year-long career!. Get gene runner manual PDF file for free from our online library. GENE RUNNER MANUAL. Gene Runner Manual Download. Gene Runner Manual Free. Gene Runner Manual Full. Gene Download gene runner 3.0; Download gene runner 3.0. Most people looking for Gene runner 3.0 downloaded: Gene Runner. Download. 4.3 on 31 votes . Gene Runner is an easy-to-use DNA

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The ANT(3’’)-Ia family of aminoglycoside nucleotidyltransferases. The gene cassette containing the aadA9 gene was present in one caprine and one bovine isolate, but the gene cassette with the aadA11 gene was the most prevalent and occurred in seven swine isolates (Table S1). Importantly, this is the first description of the aminoglycoside adenyltransferase gene cassettes aadA9 and aadA11 in T. pyogenes. Moreover, the sequences of both genes, aadA9 and aadA11, were deposited in the GenBank.Each class 1 integron always contains qacE∆1 (the quaternary ammonium compounds resistance gene), sul1 (the sulfonamide resistance gene), and orf5 (a gene of unknown function) in the 5’ conserved segment and the intI gene in the 3’ conserved segment [16]. In addition, the attI recombination site is located at the end of the 5’ conserved segment. Sequences of attI and attC, sites related to the gene cassette, may vary between different strains [28,29]. Figure 1 presents a schematic organization of the class 1 integron gene cassettes aadA11 and aadA9 detected in T. pyogenes in this study.In the investigated class 1 integron gene cassettes, both attI sites were 58 bp sequences, with the difference in one nucleotide, a cytosine in addA9 and guanine in aadA11 (Figure 2A). The attC site sequences, length 60 bp, were the same for both studied genes, and were composed of an inverse core site (GTCTAAC) and a core site (GTTAGAT) downstream of the genes (Figure 2B). 2.4. Prevalence of the Aminoglycoside Resistance Genes in Studied T. pyogenes IsolatesThe prevalence of selected aminoglycoside resistance genes among studied T. pyogenes isolates was investigated by PCR. The results are presented in Table S1 and distribution of the tested resistance genes depending on an isolate origin is shown in Table 3. The aadA9 and aadA11 genes were present in eight (9.3%) and seven (8.1%) studied isolates, respectively (Figure S1 of Supplementary Materials). The aadA11 gene was detected only in T. pyogenes swine isolates (7/21), and in all cases was carried on the class 1 integron gene cassettes. However, the aadA9 gene only in two isolates, one bovine and one caprine, was located on the class 1 integron